Search results for "Amino acid metabolism"

showing 10 items of 16 documents

Defense Priming in Nicotiana tabacum Accelerates and Amplifies ‘New’ C/N Fluxes in Key Amino Acid Biosynthetic Pathways

2020

: In the struggle to survive herbivory by leaf-feeding insects, plants employ multiple strategies to defend themselves. One mechanism by which plants increase resistance is by intensifying their responsiveness in the production of certain defense agents to create a rapid response. Known as defense priming, this action can accelerate and amplify responses of metabolic pathways, providing plants with long-lasting resistance, especially when faced with waves of attack. In the work presented, short-lived radiotracers of carbon administered as 11CO2 and nitrogen administered as 13NH3 were applied in Nicotiana tabacum, to examine the temporal changes in &lsquo

0106 biological sciences0301 basic medicineNicotiana tabacumamino acid metabolismPlant Science01 natural sciencesplant insect herbivorySerine03 medical and health scienceschemistry.chemical_compoundBiosynthesislcsh:Botanynitrogen-13Shikimate pathwaycarbon-11Secondary metabolismEcology Evolution Behavior and SystematicsX-ray fluorescence imagingchemistry.chemical_classificationEcologybiologydefense primingJasmonic acidfungifood and beveragesbiology.organism_classificationlcsh:QK1-989Amino acidMetabolic pathway030104 developmental biologychemistryBiochemistryisotope ratio analysis010606 plant biology & botanyPlants
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Insulin resistance is associated with altered amino acid metabolism and adipose tissue dysfunction in normoglycemic women

2016

AbstractInsulin resistance is associated adiposity, but the mechanisms are not fully understood. In this study, we aimed to identify early metabolic alterations associated with insulin resistance in normoglycemic women with varying degree of adiposity. One-hundred and ten young and middle-aged women were divided into low and high IR groups based on their median HOMA-IR (0.9 ± 0.4 vs. 2.8 ± 1.2). Body composition was assessed using DXA, skeletal muscle and liver fat by proton magnetic resonance spectroscopy, serum metabolites by nuclear magnetic resonance spectroscopy and adipose tissue and skeletal muscle gene expression by microarrays. High HOMA-IR subjects had higher serum branched-chain …

0301 basic medicineBlood Glucosemedicine.medical_specialtySubcutaneous FatAdipose tissueGene Expression030209 endocrinology & metabolismInflammationamino acid metabolismBiology3121 Internal medicineta3111Article03 medical and health sciences0302 clinical medicineInsulin resistanceInternal medicineGene expressionmedicineHumansAmino AcidsPhosphorylationMuscle Skeletalchemistry.chemical_classificationInflammationadiposityMultidisciplinaryAnthropometryCatabolismSisätaudit - Internal medicineSkeletal muscleNaisten- ja lastentaudit - Gynaecology and paediatricsmedicine.diseaseinsuliiniresistenssi113 Computer and information sciencesAmino acidadipose tissue3141 Health care science030104 developmental biologyEndocrinologymedicine.anatomical_structurechemistryAdipose TissueBody CompositionFemaleSignal transductionmedicine.symptomInsulin ResistanceSignal TransductionScientific Reports
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Comparative column chromatographic estimations of phenylalanine in plasma, whole blood, native and paper-dried capillary blood of healthy children an…

1984

The concentration of phenylalanine in plasma, whole venous and capillary blood, and paper-dried blood of 75 probands (25 healthy adults, 27 healthy children, and 23 patients with hyperphenylalaninaemia) were measured by use of a sensitive short column chromatography method. The comparison of the values in each group of probands by several statistic methods showed an excellent correlation of the phenylalanine concentration in paper-dried whole blood to those measured in venous plasma. Evaluation of the analytical method revealed a high sensitivity and accuracy by use of a sample volume of 50 microliter. We would therefore suggest that the estimation of phenylalanine for the diagnosis and the…

AdultCapillary actionPhenylalaninePhenylalanine030204 cardiovascular system & hematologyVeins03 medical and health sciences0302 clinical medicineHyperphenylalaninemiaColumn chromatographyPlasma/Whole bloodReference Values030225 pediatricsBlood plasmaGeneticsmedicineHumansChildAmino Acid Metabolism Inborn ErrorsGenetics (clinical)Whole bloodChromatographyChromatographyChemistryVenous PlasmaMiddle Agedmedicine.disease3. Good healthCapillariesChild PreschoolJournal of inherited metabolic disease
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In Lysinuric Protein Intolerance system y+L activity is defective in monocytes and in GM-CSF-differentiated macrophages

2010

Abstract Background In the recessive aminoaciduria Lysinuric Protein Intolerance (LPI), mutations of SLC7A7/y+LAT1 impair system y+L transport activity for cationic amino acids. A severe complication of LPI is a form of Pulmonary Alveolar Proteinosis (PAP), in which alveolar spaces are filled with lipoproteinaceous material because of the impaired surfactant clearance by resident macrophages. The pathogenesis of LPI-associated PAP remains still obscure. The present study investigates for the first time the expression and function of y+LAT1 in monocytes and macrophages isolated from a patient affected by LPI-associated PAP. A comparison with mesenchymal cells from the same subject has been a…

AdultMaleCellular differentiationlcsh:MedicinePulmonary Alveolar ProteinosisBiologyMonocytesPathogenesisYoung AdultMacrophages AlveolarmedicineHumansGenetics(clinical)Pharmacology (medical)Amino Acid Metabolism Inborn ErrorsCells CulturedGenetics (clinical)Medicine(all)chemistry.chemical_classificationResearchFusion Regulatory Protein 1 Light ChainsLysinelcsh:RMesenchymal stem cellAmino Acid Transport System y+LGranulocyte-Macrophage Colony-Stimulating FactorCell DifferentiationGeneral Medicinemedicine.diseaseLysinuric protein intoleranceMolecular biologyAmino acidGranulocyte macrophage colony-stimulating factorchemistryAminoaciduriaImmunologyPulmonary alveolar proteinosismedicine.drugOrphanet Journal of Rare Diseases
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Proteomic Analysis of Saccharomyces cerevisiae Response to Oxidative Stress Mediated by Cocoa Polyphenols Extract

2020

The present study addressed the protective effects against oxidative stress (OS) of a cocoa powder extract (CPEX) on the protein expression profile of S. cerevisiae. A proteomic analysis was performed after culture preincubation with CPEX either without stress (&minus

Antioxidantmedicine.medical_treatmentSaccharomyces cerevisiaePharmaceutical Scienceantioxidant activitySaccharomyces cerevisiaeamino acid metabolismmedicine.disease_causeAmino acid metabolismAnalytical Chemistrycocoa polyphenolslcsh:QD241-441<i>Saccharomyces cerevisiae</i>03 medical and health sciencesHistone H3chemistry.chemical_compoundBiosynthesisAntioxidant activitylcsh:Organic chemistryprotein identificationDrug DiscoverymedicineDeletion mutantsoxidative stressPhysical and Theoretical ChemistryReactive oxygen species metabolic process030304 developmental biologychemistry.chemical_classification0303 health sciencesbiologyCocoa polyphenolsChemistry030302 biochemistry & molecular biologyOrganic Chemistrybiology.organism_classificationYeastAmino acidBiochemistryChemistry (miscellaneous)Oxidative stressMolecular MedicineProtein identificationdeletion mutantsOxidative stressMolecules
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Human cationic amino acid transporter gene hCAT-2 is assigned to 8p22 but is not the causative gene in lysinuric protein intolerance

1997

Lysinuric protein intolerance (LPI) is a recessively inherited amino acid disorder characterized by defective efflux of cationic amino acids at the basolateral membrane of the intestinal and renal tubular epithelium. Recently, cDNAs encoding the related proteins hCAT-2A and hCAT-2B have been cloned. These two carrier proteins are most likely the product of the same gene, hCAT-2. Using the hCAT-2B cDNA, we assigned the hCAT-2 gene to chromosome 8p22. Furthermore, by linkage analysis in Finnish LPI families, we ruled out that hCAT-2B is involved in LPI disease.

Genetic LinkageBiologyGene mappingGenetic linkageComplementary DNAGeneticsmedicineHumansAmino acid transporterAmino Acid Metabolism Inborn ErrorsGeneGenetics (clinical)chemistry.chemical_classificationLysineChromosome MappingMembrane Proteinsmedicine.diseaseLysinuric protein intoleranceAmino acidchemistryBiochemistryAmino Acid Transport Systems BasicEffluxCarrier ProteinsChromosomes Human Pair 8Microsatellite RepeatsHuman Genetics
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Molybdenum Trioxide Nanoparticles with Intrinsic Sulfite Oxidase Activity

2014

Sulfite oxidase is a mitochondria-located molybdenum-containing enzyme catalyzing the oxidation of sulfite to sulfate in the amino acid and lipid metabolism. Therefore, it plays a major role in detoxification processes, where defects in the enzyme cause a severe infant disease leading to early death with no efficient or cost-effective therapy in sight. Here we report that molybdenum trioxide (MoO3) nanoparticles display an intrinsic biomimetic sulfite oxidase activity under physiological conditions, and, functionalized with a customized bifunctional ligand containing dopamine as anchor group and triphenylphosphonium ion as targeting agent, they selectively target the mitochondria while bein…

LightPhotochemistryMetal NanoparticlesGeneral Physics and AstronomyMolybdenum trioxidechemistry.chemical_compoundSulfiteSulfite oxidaseElectrochemistryNanotechnologyGeneral Materials ScienceBifunctionalAmino Acid Metabolism Inborn ErrorsElectrodesSulfite oxidase deficiencyMolybdenumchemistry.chemical_classificationPhotonsBinding SitesNanowiresSulfite OxidaseGeneral EngineeringOxidesAmino acidKineticsEnzymechemistryBiochemistryNanoparticlesEnzyme mimicElectronicsZinc OxideOxidation-ReductionACS Nano
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Abnormal Hypermethylation at Imprinting Control Regions in Patients with S-Adenosylhomocysteine Hydrolase (AHCY) Deficiency

2016

S-adenosylhomocysteine hydrolase (AHCY) deficiency is a rare autosomal recessive disorder in methionine metabolism caused by mutations in the AHCY gene. Main characteristics are psychomotor delay including delayed myelination and myopathy (hypotonia, absent tendon reflexes etc.) from birth, mostly associated with hypermethioninaemia, elevated serum creatine kinase levels and increased genome wide DNA methylation. The prime function of AHCY is to hydrolyse and efficiently remove S-adenosylhomocysteine, the by-product of transmethylation reactions and one of the most potent methyltransferase inhibitors. In this study, we set out to more specifically characterize DNA methylation changes in blo…

Male0301 basic medicineMethyltransferaselcsh:MedicineArtificial Gene Amplification and ExtensionGlycine N-MethyltransferaseBiochemistryPolymerase Chain Reactionlaw.inventionMethionine0302 clinical medicinelawAmino Acidslcsh:SciencePolymerase chain reactionGeneticsDNA methylationMammalian GenomicsMultidisciplinaryOrganic CompoundsGenomicsMethylationChromatinEnzymes3. Good healthNucleic acidsChemistryPhysical SciencesDNA methylationEpigeneticsFemaleDNA modificationChromatin modificationResearch ArticleChromosome biologyCell biologyAlu elementBiologyResearch and Analysis MethodsGenomic Imprinting03 medical and health sciencesAlu ElementsGeneticsSulfur Containing Amino AcidsHumansRepeated SequencesMolecular Biology TechniquesMolecular BiologyAmino Acid Metabolism Inborn ErrorsGeneBiology and life sciencesOrganic Chemistrylcsh:RChemical CompoundsInfant NewbornProteinsInfantDNAMethyltransferasesCreatineMolecular biologyLong Interspersed Nucleotide Elements030104 developmental biologyDifferentially methylated regionsAnimal GenomicsEnzymologyAHCY ; Hypermethylationlcsh:QGene expressionGenomic imprinting030217 neurology & neurosurgeryDevelopmental BiologyPLOS ONE
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Adipose Tissue Dysfunction and Altered Systemic Amino Acid Metabolism Are Associated with Non-Alcoholic Fatty Liver Disease

2015

Article

MaleMagnetic Resonance Spectroscopymedicine.medical_treatmentBiopsyAdipose tissuelcsh:MedicineFats0302 clinical medicineNon-alcoholic Fatty Liver DiseaseMetabolitesTerveystiede - Health care scienceSkeletal musclesAmino Acidslcsh:ScienceNon-U.S. Gov'tchemistry.chemical_classification0303 health sciencesMultidisciplinaryResearch Support Non-U.S. Gov'tFatty liverFatty AcidsrasvamaksaMiddle Aged3. Good healthAmino acidmedicine.anatomical_structureAdipose TissueLiverFemaleResearch Articlemedicine.medical_specialtyAdipose tissue030209 endocrinology & metabolismamino acid metabolismBiologyResearch Support03 medical and health sciencesInsulin resistanceInternal medicineFatty livermedicineJournal ArticleHumansObesityFatty acids030304 developmental biologyfatty liverCatabolismInsulinta1184lcsh:RFatty acidSkeletal muscleta3121medicine.diseaseEndocrinologychemistrylcsh:QGene expression
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Newborn screening and disease variants predict neurological outcome in isovaleric aciduria.

2021

Isovaleric aciduria (IVA), a metabolic disease with severe (classic IVA) or attenuated phenotype (mild IVA), is included in newborn screening (NBS) programs worldwide. The long-term clinical benefit of screened individuals, however, is still rarely investigated. A national, prospective, observational, multi-center study of individuals with confirmed IVA identified by NBS between 1998 and 2018 was conducted. Long-term clinical outcomes of 94 individuals with IVA were evaluated, representing 73.4% (for classic IVA: 92.3%) of the German NBS cohort. In classic IVA (N = 24), NBS prevented untimely death except in one individual with lethal neonatal sepsis (3.8%) but did not completely prevent si…

MalePediatricsmedicine.medical_specialtyAdolescentNeurocognitive DisordersDisease03 medical and health sciencesYoung AdultCognitionNeonatal ScreeningMaintenance therapyGermanyGeneticsmedicineHumansProspective StudiesMetabolic diseaseChildAmino Acid Metabolism Inborn ErrorsGenetics (clinical)030304 developmental biology0303 health sciencesNewborn screeningNeonatal sepsisIsovaleryl-CoA Dehydrogenasebusiness.industry030305 genetics & heredityInfant NewbornInfantmedicine.diseasePrognosisIsovaleric AcidemiaPhenotypeChild PreschoolCohortFemalesense organsbusinessNeurocognitiveJournal of inherited metabolic diseaseREFERENCES
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